By Teresa Conrick
I do a lot of research and writing about Autism and the Microbiome. A rising issue that encompasses this area of research is ANTIBIOTICS. I have a daughter who regressed into AUTISM and subsequently developed an AUTOIMMUNE disorder. Her blood testing showed a positive antinuclear antibody response. Right now, it is called PANS/PANDAS (Pediatric Acute-onset Neuropsychiatric Syndrome), like many other children both with and without Autism, who develop these horrific symptoms. Megan was on ANTIBIOTICS frequently as a toddler but it was not until AFTER VACCINATION that antibiotics came into the picture. She began to receive numerous VACCINES in 1993 and after that, her body seemed to change and become a sponge to pathogens. Her doctors note when Meg was 15 months old, ten days after her MMR vaccine, "6/28/94 - Fever- rash on body - ear infection - antibiotics". Many vaccines in-between antibiotics, most with Thimerosal, and regression into Autism. My daughter did not suffer an acute reaction, ie seizures immediately after a vaccine that led to the emergency room, but suffered continual regression and a very evident reaction after her MMR. The doctor's office told me this was normal... a viral infection, but then came the symptoms - loss of eye contact, inability to climb stairs, loss of language as the weeks went by. The severe GI issues then followed. Seizures came years later. Then autoimmunity. Her condition could best be described as being a type of encephalitis that at times seems to be an autoimmune encephalitis. Could it be that combining VACCINES, MERCURY and ANTIBIOTICS in a short time period is just too much for the MICROBIOME to handle? -- The effect of sole antibiotics on an infant has been studied but not with vaccinations, or environmental sources, ie mercury/Thimerosal:
...when early fecal samples from antibiotic-exposed infants were compared with a later post-weaning sample, antibiotic resistance was reduced, and overall diversity had increased . This may have been due to the plasticity and rapid rate of change within the gut microbiota in the first year of life, suggesting that effects of early antibiotic usage in infants may be diminished over time ....
...In two studies, a large fraction of healthy, non-antibiotic-treated infants in the first 3 months of life harbored resistant and multiply resistant bacterial strains [59,60], perhaps through maternal transmission . Although it has yet to be evaluated epidemiologically, the growing presence of resistant microbes may be due in part to more widespread contaminant exposures from foods and the environment. For instance, several studies demonstrated that individuals exposed to mercury were more likely to possess resistance to multiple antibiotics, suggesting a coselection mechanism . Children are regularly exposed to arsenic, which can be found in well water and foods, such as rice and baby formula [63,64,65]. Metals, such as arsenic, which was used historically as an antibiotic in humans  and is currently added to animal feed, have contributed to the emergence of metal/antibiotic coresistant strains arising in livestock, including MRSA isolates  transmittable to humans via the environment and food supply. These multiresistant pathogens heighten risk of adverse outcomes, especially in young children. Once antibiotic resistance genes are selected for, they may persist within the microbiota for years .