While channel checking one recent night, I became interested in a story about a man who seemed to have had a slowing down of his Alzheimer's symptoms by adding coconut oil into his daily diet. Many doctors who work with patients who have an autism diagnosis and immune dysfunction, have been recommending coconut oil for a number of years and it is also helping in other immune system disorders. It was this video that over five million viewers watched:
"Out of all the videos viewed on our CBN News website last year, the most popular by far showed how coconut oil helps some Alzheimer's patients. More than 5 million people watched it.
Now, some heavy-hitters within the Alzheimer's medical establishment are also taking notice. The video told the story of Steve Newport, whose Alzheimer's symptoms reversed after he started taking coconut oil.
His wife, Dr. Mary Newport, a Florida neonatologist, documented her husband's astounding progress in her book, Alzheimer's Disease: What If There Was A Cure?
......Here's how it appears the coconut oil works. In patients with Alzheimer's, insulin resistance prevents their brain cells from accepting glucose, their primary fuel. Without it, the cells die. But there is an alternate fuel known as ketones, which cells easily accept. Ketones are metabolized in the liver after eating coconut oil..........Insulin resistance doesn't just affect people with Alzheimer's but also folks with dementia, multiple sclerosis, diabetes, Parkinson's, ALS, even autism."
"Dietary protocols that increase serum levels of ketones, such as calorie restriction and the ketogenic diet, offer robust protection against a multitude of acute and chronic neurological diseases. The underlying mechanisms, however, remain unclear. Previous studies have suggested that the ketogenic diet may reduce free radical levels in the brain. Thus, one possibility is that ketones may mediate neuroprotection through antioxidant activity....Ketones also significantly decreased mitochondrial production of reactive oxygen species and the associated excitotoxic changes by increasing NADH oxidation in the mitochondrial respiratory chain.... ketones reduce glutamate-induced free radical formation by increasing the NAD+/NADH ratio and enhancing mitochondrial respiration in neocortical neurons. This mechanism may, in part, contribute to the neuroprotective activity of ketones by restoring normal bioenergetic function in the face of oxidative stress."
"The peptide beta amyloid has long been thought to be involved in Alzheimer's disease, though there is a great deal of controversy about whether it's a primary cause of the disease, or merely a symptom. Now, Rudolph Tanzi and his group at Massachusetts General Hospital have shown it might not be simply 'junk' - it could be being made to protect the brain from pathogens as part of the innate immune system.......beta amyloid has a normal function, but if there is too much of that function, it becomes toxic."
Connecting dots and seeing patterns are an important part to figuring out cause and effect in so many increasing neuropsychiatric and neurodegenerative diseases. Is it then possible that coconut oil may be actually showing its most benefit by pathogen and microbial elimination? Less microbes would equal less beta amyloid and thus, increased brain functioning. We, here at Age of Autism, continue to sound the alarm on the fact that Autism for many, represents immune system damage. Is Alzheimer's the same?
Could coconut oil then be helping by slowing or stopping this process? Let's look at some research that seems to show that could be the mechanism of its success as well: A Review Of Monolaurin and Lauric Acid
"Coconut oil and certain coconut products contain approximately 50 percent lauric acid and approximately 6–7 percent capric acid......Monolaurin, a monoester formed from lauric acid (medium chain fatty acids), has profound antiviral and antibacterial activity. Research has suggested that monolaurin exerts virucidal and bactericidal effects by solubilizing the lipids and phospholipids in the envelope of the pathogen causing the disintegration of its envelope. Recent evidence has also indicated that the antimicrobial effect is related to its interference with signal transduction in cell replication6.
Lipid-Coated Bacteria Inactivated by Monolaurin Listeria monocytogenes Helicobacter pylori (gram-negative) Hemophilus influenzae (gram-negative) Staphylococcus aureus Strptococcus agalactiae Groups A, B, F, and G streptococci Gram-positive organisms Gram-negative organisms if pretreated with a chelator Lipid-Coated Viruses Inactivated by Monolaurin Human Immunodeficiency virus HIV-, HIV+ Measles Virus Herpes simplex virus-1 Herpes simplex virus-2 Herpes viridae (all) Human lymphotropic viruses (type 1) Vesicular stomatitis virus Visna virus Cytomegalovirus Epstein-Barr virus Influenza virus Pneumono virus Sarcoma virus Syncytial virus Rubeola virus
"It is noteworthy that coconut oil was active against species of Candida at 100% concentration compared to fluconazole. Coconut oil should be used in the treatment of fungal infections in view of emerging drug-resistant Candida species."
"The purpose of the present study was to evaluate the susceptibility of Helicobacter pylori to the in vitro bactericidal properties of medium-chain MGs and FFAs [free fatty acids ]."......., lauric acid (C12:0) was the only medium-chain saturated FFA with bactericidal activity against H. pylori. ...Collectively, our data demonstrate that H. pylori is rapidly inactivated by medium-chain MGs and lauric acid .."
Then there is the multitude of research showing that herpes simplex virus is connected to Alzheimer's. Here is the downstream effect to beta amyloid:
"Our research showing that herpes simplex virus type 1 (HSV1) is a risk factor for AD for the brains of people who possess a specific genetic factor and that the virus causes accumulation of key AD proteins (β-amyloid (Aβ) and abnormally phosphorylated tau (P-tau)), suggests that anti-HSV1 antiviral agents might slow AD progression.....the anti-HSV1 antiviral agents acyclovir, penciclovir and foscarnet reduced Aβ and P-tau accumulation, as well as HSV1...The antiviral-induced decrease in Aβ is attributable to the reduced number of new viruses, and hence the reduction in viral spread. Since antiviral agents reduce greatly Aβ and P-tau accumulation in HSV1-infected cells, they would be suitable for treating AD with great advantage unlike current AD therapies, only the virus, not the host cell, would be targeted."
"Steve has a long time history of lengthy outbreaks of fever blisters on his mouth, and had an outbreak around his eye and was quite sick at age 29. I strongly believe that, at least in his case, herpes simplex may be the cause of his Alzheimer's disease and this particularly bad episode could explain why he became symptomatic so early."
It appears that we are looking at immune dysfunction in many of these diseases. Like Autism, Alzheimer's has its connection to microbes . What can cause a body to not be able to fight off these pathogen predators?
"MICROBES AND IMMUNE REACTIONS- There are a number of environmental conditions that may provoke and/or weaken the immune system—infections, cancer, allergens, stress, sleep deprivation, vaccinations, trauma, toxins, degenerative changes, molecular mimicry, low glutathione levels, increased oxidative stress, metal toxicity, elevated leptin levels and some medical treatments [10-13]. "
Forget genetic connections. Go buy some coconut oil.
Teresa Conrick is Contributing Editor to Age of Autism.